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Vererbte Thrombophilie und wiederholte spontane Fehlgeburten
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ISBN10: 6208879159
ISBN13: 9786208879150
Publisher: Verlag Unser Wissen
Published: Jul 17 2025
Pages: 72
Weight: 0.24
Height: 0.17 Width: 6.00 Depth: 9.00
Language: German
ISBN13: 9786208879150
Publisher: Verlag Unser Wissen
Published: Jul 17 2025
Pages: 72
Weight: 0.24
Height: 0.17 Width: 6.00 Depth: 9.00
Language: German
Plazentatrophoblasten exprimieren Gerinnungskomponenten für die Entwicklung der Plazentagefä e. Ein Versagen dieser Funktion und die Entwicklung einer intraplazentaren Pathologie führen zu einer Plazentainsuffizienz. Thrombophilie bleibt in der Regel unentdeckt; die meisten Träger sind asymptomatisch. Wiederholte spontane Fehlgeburten (RSM) betreffen 2-5 % der Paare, wobei die Ursache in etwa 50 % der Fälle unbekannt ist. Eine Thrombose der Dezidua-Gefä e beeinträchtigt den initialen Vaskularisierungsprozess bei der Einnistung. Eine erworbene Thrombophilie (Antiphospholipid-Antikörper, Anticardiolipin-Antikörper und Lupus-Antikoagulans) tritt häufiger bei Frauen mit Antiphospholipid-Syndrom im ersten Trimester auf. Eine vererbte Thrombophilie (Faktor V (FVL), Prothrombin G20210A und Methylentetrahydrofolatreduktase (MTHFR C677T)-Genmutationen) ist ein Risikofaktor für RSM. FVL identifiziert eine aktivierte Protein-C-Resistenz mit hyperkoagulablem Zustand. Prothrombin G20210As ist mit erhöhtem Prothrombin assoziiert. MTHFR C677T verringert die MTHFR-Aktivität mit verminderter 5-Methyltetrahydrofolatsynthese, die für die Umwandlung von Homocystein in Methionin erforderlich ist, wodurch der Homocysteinspiegel im Plasma ansteigt. Unexplained RSM im ersten Trimester kann eine thrombotische Ätiologie haben; histologische Untersuchungen berichten häufig über Plazenta-Mikrothromben bei RSM.
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