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Trombofilia hereditária e abortos espontâneos repetidos
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ISBN10: 6208879191
ISBN13: 9786208879198
Publisher: Edicoes Nosso Conhecimento
Published: Jul 17 2025
Pages: 68
Weight: 0.23
Height: 0.16 Width: 6.00 Depth: 9.00
Language: Portuguese
ISBN13: 9786208879198
Publisher: Edicoes Nosso Conhecimento
Published: Jul 17 2025
Pages: 68
Weight: 0.23
Height: 0.16 Width: 6.00 Depth: 9.00
Language: Portuguese
Os trofoblastos placentários expressam componentes da coagulação para o desenvolvimento vascular placentário. A falha desses componentes e o desenvolvimento de patologias intraplacentárias garantem a insuficiência placentária. A trombofilia geralmente não é diagnosticada; a maioria dos portadores é assintomática. O aborto espontâneo recorrente (RSM) afeta 2 a 5% dos casais, com causa desconhecida em cerca de 50% dos casos. A trombose dos vasos da decidua prejudica o processo inicial de vascularização na implantação. A trombofilia adquirida (anticorpos antifosfolÃpides, anticorpos anticardiolipina e anticoagulante lúpico) é mais elevada no primeiro trimestre em mulheres com sÃndrome antifosfolÃpide. A trombofilia hereditária [fator V (FVL), protrombina G20210A e mutações do gene metilenotetrahidrofolato redutase (MTHFR C677T)] é um fator de risco para RSM. O FVL identifica a resistência à proteÃna C ativada com estado hipercoagulável. A protrombina G20210As está associada a nÃveis elevados de protrombina. O MTHFR C677T diminui a atividade do MTHFR com redução da sÃntese de 5-metiltetrahidrofolato, necessário para a conversão da homocisteÃna em metionina, aumentando a homocisteÃna plasmática. A RSM inexplicável no primeiro trimestre pode ter etiologia trombótica; estudos histológicos relatam comumente microtrombos placentários na RSM.
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